[LuvMuhRoids]

Clomid (Clomiphene Citrate), stimulates the hypophysis to release more gonadotropin so that a faster and higher release of follicle stimulating hormone aud luteinizing hormone occurs. This results in an increase of the body's own testosterone production. Clomid is a synthetic estrogen, however it does also work as an anti-estrogen. How does it work? Because it is a weak synthetic estrogen, it will bind to the estrogen receptor (ER) and not cause any problems. At the same time the increase in estrogen
from steroids are blocked from attaching to the ER.

Clomid therapy usually is administered in dosages of a 300/100/50 split. This is known as a saturation technique administering 300mg from day one to raise levels quickly. Administration is done orally at 300mg day1, 100mg 10days, 50mg 10days. Other methods have shown recovery but this seems to be the preferred method. Another beneficial technique is 100mg over a period of 30 days along with 20mg of nolvadex.

It is in my opinion, clomid is a weak anti-estrogen blocker and is a selective SERM. Clomid is selective towards hypothalamus & pituitary(also the ovary, endometrium, vagina, and cervix), which also accounts for the de-sensitization to GnRH. As opposed to nolvadex.

Clomid and Nolvadex are both anti-estrogens belonging to the same group of triphenylethylene compounds but they act very differently at different sites of action. In my opinion this is one reason why both should be used in conjuction to stimulate LH levels and restore HPTA.

It is suggestable to administer clomid therapy post cycle in different times after last injection. Reason is, androgen levels must be low enough so not to render clomid useless or inhibit its actions to stimulate LH levels. Also, If the androgen receptors are still active it sends signals through the HPTA so shut test down. Thus clomid is in a useless battle and only purpose is to serve as an anit-estrogen blocker.

Clomid stimulates the LH levels back to normal for proper restoration of the HPTA. Nolvadex along with clomid being two serms are individualy seletive to certain tissue. So it is ideal to run both. When you cease steroid administration a certain estrogen backlash will happen. This is because you have no testosterone in your system as well as the non-production of your own test. At this point estrogen floods the receptors and you will experience a crash and become subceptable to gyno symptoms or inflamation of the mammory glands.

Clomid is has anti-estrogen abilities but not nearly as strong as nolvadex thus you need them both. Important note: Clomid does not stimulate the release of natural testosterone, but rather works at reducing the oestrogenic inhibition caused by the steroid cycle.

Below is a reference chart to resort to for clomid therapy. It is suggested to run 20mg of nolvadex in conjuction with clomid therapy. This combo is ideal. Higher dosages of course are needed if gyno symptoms or inflamation of the glands appear at about 60-80mg's through out until symptoms subside.

Anadrol/Anapolan: 24 hours after last administration
Deca: 21 days after last injection
Dianabol: 24 hours after last administration
Equipoise: 21 days after last injection
Fina: 3 days after last injection
Primobolan depot: 14 days after last injection
Sustanon: 18 days after last injection
Testosterone Cypionate: 18 days after last injection
Testosterone Enanthate: 14 days after last injection
Testosterone Propionate: 3 days after last injection
Testosterone Suspension: 24 hours after last administration
Winstrol: 24 hours after last administration

LMR



CYPIONATE TIMING:
The only other suggestion is the debate when to start PCT with Cypionate. Although, almost identical to Enanthate. Cypionate has just a little more half life than Enanthate and it has been suggest to wait up to 18 days for PCT instead of 14days.

NOTE: Either way the point to clomid therapy time is to wait for the androgen levels to be low enough so clomid activity is not rendered useless. Much of the therapy time also depends on dosage, saturation, and how the user metabolizes the ester. The only true way is bloodwork.

LMR